USP maju dalam studi untuk menyesuaikan gerakan pasien dengan AME

Sebuah studi oleh Sekolah Pendidikan Jasmani dan Olahraga USP menemukan bahwa orang dengan Atrofi Otot Tulang Belakang mampu menyesuaikan gerakan tubuh mereka untuk melakukan tugas motorik. Penelitian ini dilakukan oleh Ana Angélica Ribeiro de Lima, di bawah bimbingan Profesor Edison de Jesus Manoel.

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Hasil studi

  • Studi tersebut melibatkan sembilan orang yang didiagnosis dengan AME, dengan tingkat kompetensi motorik yang berbeda.
  • Setiap empat bulan, selama satu tahun, peserta mengirimkan video yang melakukan tugas perubahan postur: dari berbaring telentang menjadi duduk, berulang kali.
  • Adaptasi diamati sehingga tugas dapat dilakukan bahkan dengan keterbatasan motorik saat ini. Mereka terutama melibatkan penggunaan tungkai atas untuk menarik diri dengan pakaian, paha atau tepi tempat tidur, atau mendorong kasur untuk membantu dalam pemindahan posisi.
  • Dari perspektif baru ini, dimungkinkan untuk mempertimbangkan adaptasi ini sebagai kemampuan organisme yang efisien dan cerdas untuk mengatasi keterbatasan yang disebabkan oleh penyakit.
  • Bagi Ana Angélica Ribeiro de Lima, penting untuk melihat badan-badan ini mampu dan kreatif, sama seperti orang-orang tanpa disabilitas.
  • “Sangat penting untuk memahami bahwa tidak ada 'cara yang benar' untuk bergerak, karena orang-orang ini memiliki variasi yang cukup besar di antara mereka sendiri dan menemukan strategi yang berbeda untuk menyelesaikan masalah motorik. Selain itu, para profesional dapat memperoleh manfaat dari memiliki akses ke deskripsi terperinci dari berbagai strategi ini, untuk menerapkannya dalam perawatan pasien mereka”, peneliti menyimpulkan.
Sebuah penelitian menunjukkan bahwa adaptasi gerakan dapat mengatasi keterbatasan yang disebabkan oleh penyakit (Gambar: Reproduksi / EEFE)

Apa itu Atrofi Otot Tulang Belakang?

  • Spinal Muscular Atrophy (AME) adalah penyakit progresif yang gejala utamanya adalah kelemahan otot.
  • Ini dapat menyebabkan masalah mobilitas, pernapasan, bicara dan menelan.
  • Penyebab penyakit ini turun-temurun dan terkait dengan cacat pada gen yang menghasilkan protein SMN, yang bertanggung jawab atas kelangsungan hidup neuron motorik.

Dengan informasi dari Journal of USP.

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